Showing posts with label Scholarly articles on cardiology. Show all posts
Showing posts with label Scholarly articles on cardiology. Show all posts

Monday, June 24, 2019

Lupine Publishers - Journal of Cardiology Research


Psychopathy of the careerist is a developmental disorder marked by emotional deficits and an increased risk for antisocial behavior. It is not equivalent to the diagnosis Antisocial Personality Disorder, which concentrates only on the increased risk for antisocial behavior and not a specific cause-ie, the reduced empathy and guilt that constitutes the emotional deficit. Our review considers data regarding the neurobiology of this disorder. Dysfunction within the amygdala’s role in reinforcement learning and the role of ventromedial frontal cortex in the representation of reinforcement value is stressed. Data is also presented indicating potential difficulties within parts of temporal and posterior cingulate cortex. Suggestions are made with respect to why these deficits lead to the development of the disorder. Knowledge of recent neuorobiology is proving our thesis that Darwin was wrong when formulated his theorem„ Survival of the fittest“. Reality in 21 century is showing that “Survival of the careerist“ based on the Quantum Entanglement Entropy (QEE) is more valid principle of Social Dynamics in our days. Careeristic Competition is the main cause of the QEE leading to icreased complications through Coincidenses of Social Dynamics - Lupine Publishers.

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Wednesday, April 17, 2019

Scholarly Articles on Cardiology- Lupine Publishers



Does Raising the Central Venous Pressure (CVP) in Treating Shock with Fluids Induce Volumetric Overload Shocks (VOS)? by  Ahmed N Ghanem in ACR - Lupine Publishers


The answer to the heading question is an affirmative yes! Not only persistence to elevate the central venous pressure (CVP) but also pulmonary capillary wedge pressure (PCWP) is among the fundamental commonly practiced but most misleading reasons for overzealous fluid infusion in managing haemodynamic shock of poly-trauma victims and inducing volumetric overload shock (VOS). The generally received practice on raising CVP and PCWP to level of 18-22 cm water in the management of shock as well as in acutely ill patients is a misconception [1]. All physiology textbooks testify that the normal CVP is 0 and ranges between -7 to +7 cm water measured at middle auxilary line- the level of right atrium on supine posture [2]. The best advice here is that we should imitate nature exactly if we can’t understand how it truly works. Raising CVP to such high level vastly increases filtration force at capillaries inducing VOS and causing oedema of the multiple vital organ dysfunction/ failure (MVOD/F) as detailed in the articles [3,4]- Lupine Publishers.


https://lupinepublishers.com/cardiology-journal/fulltext/does-raising-the-central-venous-pressure-cvp-in-treating-shock-with-fluids-induce-volumetric-overload-shocks-vos.ID.000121.php
https://lupinepublishers.com/cardiology-journal/pdf/ACR.MS.ID.000121.pdf 

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Wednesday, January 30, 2019

Cardiology-journal_Lupine Publishers



Plenty has been written about the impact of arteriosclerosis, i.e. arterial biomineralization, on the functioning of human body. A little less information is available concerning the dissolution and prevention of such mineralization (calcium channel blockers etc.) [1,2]. The significance of even a small progress in “cleaning” the arteries cannot be overstated. By renewing the proper functioning of the cardiovascular system, it will restore proper functioning of tissue, organs, and other elements of the body, and in effect support removal or reduction of many diseases. It will lead to lasting rejuvenation of the body.


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Interruption of the Aortic Arch in the Adult and Fulminant Myocarditis: A Strange Presentation

Introduction   53 years old female patient, who presented oppressive precordial pain, radiating to the neck and jaw, for which she went to...